Roidipedia.Compound reference & reporting
05 AUG 2026
CompoundsPharmaceutical Prednisolone
PharmaceuticalGlucocorticoidCorticosteroidAnti-inflammatory

Prednisolone

Also known as Prelone · Orapred · Millipred

Prednisolone is the active glucocorticoid to which prednisone is converted in the liver, so it does not require hepatic activation and is preferred where liver conversion may be impaired. It shares prednisone's roughly fourfold anti-inflammatory potency over cortisol and the same double-edged profile: excellent inflammation control but catabolic, immunosuppressive and HPA-suppressive with sustained use.

01 Overview

Because prednisolone is already the active molecule, it is used interchangeably with prednisone in most settings and is favoured in liver disease or when a rapidly reliable systemic effect is wanted. It is available orally, intravenously and as eye drops. Its glucocorticoid receptor activity, indications and adverse-effect spectrum mirror prednisone almost exactly.

For athletes the same caveats apply. Systemic prednisolone opposes muscle anabolism, drives central adiposity, weakens bone and suppresses immunity and the HPA axis. It is prohibited in-competition by WADA via oral, intravenous, intramuscular and rectal administration.

02 Mechanism

Active glucocorticoid that binds the glucocorticoid receptor to suppress inflammatory transcription and drive gluconeogenesis and protein catabolism; no hepatic activation required.

03 Dosing

TierDoseRouteNotes
Short burst20–60 mg/dayOralAnti-inflammatory burst; brief courses usually need no taper.

04 Effects

EffectMagnitudeEvidence
Anti-inflammatory actionDirectly active without needing hepatic conversion; reliable inflammation control.rapid, broadClinical
Muscle catabolismSustained use promotes muscle protein breakdown and steroid myopathy.dose/duration dependentClinical

05 Side effects

EffectSeverityFrequencyEvidenceCountermeasures
HPA axis suppressionSuppresses endogenous cortisol; abrupt withdrawal after prolonged courses risks adrenal insufficiency.SevereUniversal with prolonged useClinical
ImmunosuppressionRaised infection risk and impaired healing.SevereCommon with doseClinical
Fluid retention and hypertensionSodium/water retention raising blood pressure.ModerateCommonClinical

07 References

Systemic glucocorticoids: pharmacology and adverse effectsAnn Rheum Dis, 2016
WADA Prohibited List - glucocorticoidsWorld Anti-Doping Agency

08 Discussion0 comments

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This page is reference information, not medical advice. Doses and protocols are documented as they appear in the clinical literature and in practice — describing them is not a recommendation to use them. Countermeasures listed here are not a substitute for a physician. Legal status varies by jurisdiction and changes.