Roidipedia.Compound reference & reporting
06 AUG 2026
CompoundsSARM Ligandrol (LGD-4033)
SARMNon-steroidalOralSARM

Ligandrol (LGD-4033)

Also known as LGD-4033 · VK5211 · Anabolicum

Ligandrol (LGD-4033) is a non-steroidal SARM with higher potency than ostarine, studied in small Phase I trials for safety and lean-mass effects. It is popular in anecdotal recreational use for strength and size but reliably suppresses testosterone even at low milligram doses. It is unapproved, prohibited in sport, and linked to case reports of liver injury.

01 Overview

LGD-4033, developed by Ligand Pharmaceuticals and later Viking Therapeutics, showed dose-dependent increases in lean body mass and dose-dependent testosterone suppression in a three-week Phase I study in healthy men. Its higher androgen-receptor affinity means effects and suppression appear at lower doses than ostarine.

Clinical data remain limited to early-phase safety work; no efficacy trials support recreational claims. Ligandrol is a recurrent cause of anti-doping violations and appears in case reports of hepatotoxicity. Like other SARMs it is sold only as an unregulated research chemical.

02 Mechanism

High-affinity, tissue-selective androgen-receptor agonist that promotes anabolic signalling in muscle and bone with reduced action on androgenic tissues.

03 Dosing

TierDoseRouteNotes
Light2–5 mg/dayOral1 mg produced measurable lean-mass gain in trials; recreational use often exceeds this.
Common5–10 mg/dayOralAnecdotal range; suppression scales with dose.

Ester comparison

EsterHalf-lifeInjection freq.Testosterone by mass

04 Effects

EffectMagnitudeEvidence
Increased lean body massA Phase I study found dose-dependent lean-mass increases over placebo in healthy young men.~+1.2 kg over 3 weeks at 1 mgClinical
Increased strengthUsers report notable strength gains; not quantified in controlled trials.Anecdotal

05 Side effects

EffectSeverityFrequencyEvidenceCountermeasures
Testosterone suppressionDose-dependent suppression of total testosterone, SHBG, LH and FSH observed even at 1 mg; recovery within weeks of stopping in the trial.ModerateNear-universalClinical
HDL cholesterol reductionTransient dose-dependent decreases in HDL reported in trials.ModerateCommonClinical
HepatotoxicityCase reports describe cholestatic or hepatocellular liver injury after LGD-4033 use.ModerateRareObservational

06 Commonly used with

What ligandrol is combined with, and why. This is about intent rather than safety — the interactions table below covers what is dangerous. Nothing here is listed without what it costs.

CompoundFrequencyPurpose & trade-off
Stacked compounds
OstarinerecompCommonOstarine is stacked in as a milder muscle-preservation agent so the pair covers both size and conditioning during a lean bulk or recomp.Trade-off: Two suppressive SARMs together deepen the hormonal shutdown beyond either alone, making a proper PCT more necessary.
CardarineenduranceCommonA non-hormonal endurance and fat-loss add-on that offsets the sluggish conditioning some report on a ligandrol bulk.Trade-off: Cardarine's rodent carcinogenicity signal and absence of long-term human data mean it adds its own risk rather than being a free benefit.
Support & ancillaries
TestosteroneTRT baseOccasionalSome run a TRT-dose testosterone base to keep androgen levels and libido functional given ligandrol's suppression.Trade-off: This converts a nominally oral-only cycle into an injectable steroid cycle, adding aromatisation, blood-pressure and full-suppression concerns.
Post-cycle
ClomifenePCTCommonUsed as post-cycle therapy to restart natural testosterone, as ligandrol is reliably suppressive even at moderate doses.Trade-off: Clomifene frequently causes mood swings and visual disturbances, and recovery is not assured after longer or higher-dose runs.

08 References

Safety, pharmacokinetics and pharmacodynamics of LGD-4033 in healthy menJ Gerontol A Biol Sci Med Sci, 2013
Ligandrol clinical literaturePubMed

09 Discussion0 comments

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This page is reference information, not medical advice. Doses and protocols are documented as they appear in the clinical literature and in practice — describing them is not a recommendation to use them. Countermeasures listed here are not a substitute for a physician. Legal status varies by jurisdiction and changes.